There is skin that ages in tune with the calendar — gradually, calmly, as if following an inner natural metronome. And there is another type — skin that suddenly looks older than the date in the passport suggests: dull, slightly puffy in the morning, less responsive to creams that used to work perfectly. It reacts faster, tires sooner and flushes from touches that caused no reaction just yesterday.

Such skin is often described as “tired”, but this word is too superficial. In reality, it lives in a state of silent internal inflammation — barely noticeable yet constant, like a low background noise the body has learned to ignore, while the skin is still trying to speak about it. This explains why a face that looked “normal” only recently can suddenly appear thinner, less resilient and sensitive to any change in skincare.

This process has a name familiar to immunologists and gerontologists: inflamaging — a silent, low-grade inflammation that accumulates over time and changes the pace of skin aging. And the more carefully we look at the skin not only in the mirror but also through the lens of biochemistry, the clearer it becomes: this is not a “whim”, but a language the body uses to talk about exhaustion.

What is inflamaging — and why it feels like a background state rather than a symptom

Classic acute inflammation is easy to recognize: heat, redness, swelling, clear discomfort. It is a bright, short-term reaction of the immune system that raises an alarm and then subsides once the threat has been resolved.

Inflamaging behaves differently. It is not a blaze but a slow smoldering. There is no sharp pain, no obvious trigger, no reaction that forces us to take immediate action. Instead, there is a constant presence — a low level of inflammation that alters cellular behavior and gently disturbs the usual “musical score” of the skin.

On a cellular level, it looks like this: immune cells shift into a semi-active state, as if they are always waiting for a signal; they release small portions of cytokines — not enough to cause acute pain, but sufficient to influence the tissues. Fibroblasts work in a less stable mode, collagen is broken down faster than it is synthesized, new fibers form more fragile, the barrier gradually loses density, and the skin starts reacting to stimuli that previously went unnoticed.

The rhythm of inflamaging is amplified by the same processes that change our hormonal balance during stress or travel. In our article on hormonal changes on the road, we explained in detail how such fluctuations affect skin reactivity: https://union.beauty/publications/travel-that-changes-hormones/.

Where this silent internal inflammation comes from

Inflamaging almost never has a single cause. It appears where many small irritants work together at the same time — often for years. It can be irregular sleep that disrupts circadian rhythms, chronic fatigue, work overload, constant emotional tension, a diet rich in ultra-processed foods, polluted city air, dry indoor environments, UV exposure even on cloudy days, constant air conditioning and sharp temperature changes.

All of this creates a background in which the skin no longer has time to fully recover. It cannot “close” small micro-damages before new ones appear on the horizon. At some point we no longer see separate reactions but a general picture: the face seems to have lost its ability to return to a state of calm.

Increased skin reactivity often mirrors what happens in the nervous system: emotional fluctuations, changes in muscle tone and chronic inner tension can all reinforce low-grade inflammation. We explored this interaction in detail in our article on sensitive skin and emotions: https://union.beauty/publications/when-the-body-is-tired-of-emotions-what-the-skin-feels/.

How inflamaging changes the structure of the skin

If we look at the skin not only in the mirror but also in terms of its structure, it becomes clear why silent inflammation has such a visible impact on appearance. Under the influence of chronic low-grade inflammation, enzymes that break down collagen and elastin become more active, new fibers form less elastic and more fragile, and the lipid barrier gradually thins.

At this point, transepidermal water loss (TEWL) increases: the skin becomes less able to retain moisture, dries out more quickly, and is more sensitive to changes in temperature and humidity. Any familiar product that used to feel comfortable can suddenly cause tightness, tingling or mild burning. At the same time, inflammatory processes may influence pigmentation: post-inflammatory spots linger longer, the tone looks uneven, and even minor breakouts take more time to heal.

This is how a familiar feeling arises: “my skin has become thin and tired, it reacts to everything”. In reality, it is not so much the “personality” of the skin that changes, but the background in which it exists: what once was a temporary reaction gradually becomes its default mode.

Close-up of natural facial skin texture with subtle micro-relief and gentle reactivity, characteristic of silent inflammation.

Can we “switch off” inflamaging?

It is neither possible nor necessary to completely switch off inflammatory processes in the body. Inflammation itself is an essential part of the defence and repair systems; without it, the skin would not be able to heal. The goal in skincare and lifestyle is different: not to make the skin “sterile and calm”, but to reduce background tension, remove unnecessary triggers and restore the tissue’s ability to recover in its natural rhythm.

In practice, this means three key steps. First, to gently reduce the number of stimuli that keep “feeding the fire”: rethinking skincare, lighting, microclimate, sleep routine and nutrition. Second, to support the barrier and microbiome so that the skin can cope better with unavoidable external factors. Third, to choose actives that work gently, without overloading a system that is already exhausted.

The baseline level of inflammation decreases when the body returns to a calmer rhythm — with pauses, rest, warm rituals and space for recovery. We explored this mechanism in our article on the therapeutic role of silence and its effect on the nervous system: https://union.beauty/publications/sensory-detox-when-the-mind-hears-silence/

How skincare can help the skin leave the state of chronic defence

The first step is to honestly look at your routine. If it includes several types of acids, retinoids, frequent peels and harsh cleansers, while the skin is dry, irritated and often red, it is highly likely that it is not “picky” but overloaded. In this state, any additional strong active works not as a helper but as one more trigger.

The logical move is a temporary shift towards the “less but wiser” formula. This means gentle yet effective cleansing that does not strip the skin, formulas with repairing components (ceramide-like structures, fatty acids, soothing extracts), fermented complexes and probiotic fractions, a controlled amount of anti-inflammatory actives, and daily sun protection — even on cloudy days or when working indoors by a window.

It is also important to pay attention to textures. For skin with signs of inflamaging, not only the ingredient list matters but also how the product feels: whether there is immediate tightness after application, whether there is burning, how quickly comfort returns. Often it is these micro-signals that best show whether the barrier is ready for the next step or still needs time to recover.

The role of fermented skincare in the context of silent inflammation

Fermentation in cosmetics is not just a trendy slogan, but a way to affect the skin more delicately. Fermented extracts and probiotic complexes work with two key links in inflamaging: the microbiome and the barrier function. Optimised molecular size and bioactive components allow them to act effectively at lower concentrations, reducing the risk of aggressive impact.

Supporting the microbiome helps beneficial microorganisms maintain balance on the skin surface, reducing its tendency towards chronic inflammation. Strengthening the barrier, in turn, allows the skin to retain moisture more effectively, respond more calmly to changes in temperature and humidity, and better tolerate the inevitable stresses of modern life. Where once we had to constantly “put out fires”, we now gain the opportunity to work preventively.

When “heavy artillery” products in a routine are replaced with formulas that soothe, repair and gently correct at the same time, the skin gradually leaves the state of permanent defence. This is not instant magic and not an overnight effect, but exactly the pace that a system, which has spent years in a state of silent inflammation, needs.

What is worth remembering

Inflamaging is not a sentence and not a reason to fear every patch of redness. It is a useful concept that helps structure our experience: to understand why the skin sometimes looks older than we expect, why it tires from too many actives and why “one more serum” is not always the answer.

When we look at skincare through the lens of silent inflammation, decision-making becomes easier: remove the excess, protect the barrier, support the microbiome, choose non-traumatising textures. And then — patiently observe how the skin gradually stops living in a state of war with the outside world and returns to its natural, calmer and more youthful appearance.

References

  • Pan, T. et al. (2023). Aging and chronic inflammation: highlights from a multidisciplinary perspective. Immunity & Ageing.
  • Zhuang, Y., & Lyga, J. (2014). Inflammaging in skin and other tissues – the roles of complement system and macrophage. Inflammation & Allergy – Drug Targets.
  • Cheng, B., Yang, J., Song, Y., Zhang, D., & Hao, F. (2022). Skin immunosenescence and type 2 inflammation: a mini-review with an inflammaging perspective. Frontiers in Cell and Developmental Biology.
  • Rodriguez, E. et al. (2023). The skin and inflamm-aging. Biology (Basel).